https://ogma.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:50452 Wed 26 Jul 2023 13:14:54 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:23815 Wed 11 Apr 2018 09:44:11 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:19484 Wed 04 Apr 2018 17:03:10 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:34168 Mon 18 Feb 2019 13:59:08 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:50096 1c 8.4 ± 1.6%) were included in this cross-sectional analysis. Carotid-femoral pulse wave velocity (cfPWV) was used to quantify arterial stiffness. Aerobic fitness (relative V̇O_2peak ) was determined via indirect calorimetry during maximal exercise testing. Central body fatness was determined using waist circumference. Data were analysed using hierarchical multiple regressions. After adjustment for sex and duration of T2D, each one standard deviation (SD) increase in age (8.68 years) was associated with a 0.63 m·s^-1 increase in cfPWV (β = 0.416, p = 0.001). Following adjustment for aerobic fitness and body fatness, the standardized β was unchanged (0.417). A one SD increase in waist circumference (13.9 cm) and relative V̇O_2peak (5.3 ml·kg^-1 ·min^-1 ) were associated with a similar magnitude of difference in cfPWV (0.47 m·s^-1 and -0.44 m·s^-1 , respectively). Therefore, age is a significant correlate of increased arterial stiffness in T2D, with higher aerobic fitness attenuating, and higher body fatness exacerbating, this increase. Interventions aimed at improving cardiovascular outcomes in people with T2D should target both increased aerobic fitness and reduced body fatness.]]> Mon 17 Jul 2023 11:57:25 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:40059 Fri 22 Jul 2022 10:05:54 AEST ]]>